Below: This anabolic steroid pumped body is only for show.
Below: This photo was taken at the age of 64. I can whip his fellow's ass on 25 kg dumb bells any time -- I will lift double in half the time
Below:This photo was taken at the age of 64. I can whip his fellow's ass on 25 kg dumb bells any time -- I will lift double in half the time
Below: In the videos below, I just stopped as soon as a wager was done with.
I could have continued -- for how long?-- that is a secret .
Never show all -- your armory.
THIS IS ME AT THE AGE OF 61
Below: Eddie Hall lifts 500 Kilos and sets world record.
This is NOT a legal lift as he used wrist straps -- which basically fools the brain into lifting more.
Fingers MUST convey signals to the brain.
He wore tendon supporters under his T shirt
But when you do that you endanger your own body
Eddie Hall's ligaments could be heard popping by people around him. His whole body became so HOT and PAIN WRACKED that they had to keep him in ice.
He suffered severe anterior and posterior nose bleeds and this could have killed him.
Posterior nosebleeds usually involve larger blood vessels than anterior nosebleeds. In posterior nosebleeds, the bleeding originates further back and higher up the nose in an area where artery branches supply blood to the nose; this is why the bleeding is heavier.
Posterior nosebleeds are often more serious than USUAL anterior nosebleeds..
Posterior bleeds usually drain down the back of the throat, but can also bleed out of both nostrils.
Swallowed blood may pass through the digestive tract and appear in the stool as black tarry stools.
When you lift extra heavy weights , your heart pumps blood throughout your body at a greater rate as your muscles demand extra oxygen. Some of that blood flows through the little capillaries inside the nose itself. It doesn't take much to damage these capillaries.
Below: This lifter has strong tendons and ligaments ..
The Valsalva maneuver is the voluntary manipulation of air in the body to create increased tension and pressure during lifts.
When a STUPID coach instructs you to breathe in and hold your air throughout a lift, then you’re performing the Valsalva maneuver. This style of breathing increases blood pressure and tension on the body by preventing oxygen to flow as freely.
Many weightlifters have died on stage because of hyperventilation followed by Valsava manoevre .
Ligaments join one bone to another bone, while tendons connect muscle to bone.
CUNTS WHO CONSUME ANABOLIC STEROIDS DO NOT HAVE STRENGTH — THEIR HUGE MUSCLES ARE FOR SHOW , LIKE PLASTIC BOOBS OF WOMEN
Users of anabolic steroids are more likely to lift heavier weights than non users to impress others —because these IDIOTS feel that BIG muscles make you strong
BELOW: OH BABY -YOU SET MY ASS --NAY -- SOUL ON FIRE !
BABY, CAN YOU WHISTLE WITH YOUR TWAT ?
BELOW: OH BABY -YOU SET MY ASS --NAY -- SOUL ON FIRE !
BABY, CAN YOU WHISTLE WITH YOUR TWAT ?
TENDONS ARE DEGRADED BY ANABOLIC STEROIDS ..
CAPT AJIT VADAKAYIL TELLS WHAT EVIL PHARMA BRIBED DOCTORS DO NOT TELL YOU
TO SHOW OFF, MEN WHO HAS STEROID PUMPED HUGE MUSCLES , LIFT MORE WEIGHTS THEN THEIR TENDONS CAN HANDLE.. THEY RUPTURE THEIR TENDONS
More than 93% of tendon injuries are long term in nature
Anabolic androgenic steroids (AAS) are highly potent drugs, with “anabolic” meaning “to grow”..
AAS are a class of synthetic drugs that mimic the effects of the body’s naturally occurring hormone, testosterone. Use of AAS among teenagers and males in their early 20s who are still growing cause stunted bone growth.
WARNING: Anabolic steroids give large muscles - BUT they weaken the tendons .
Anabolic steroid use results in dysplasia of collagen fibrils, causing a drastic reduction in the overall tensile strength of the body’s tendons. AAS use makes you susceptible to tendon rupture
YOU CAN HEAR YOUR TENDON SNAP
The forces applied to a tendon may be more than 5 times your body weight.
A tendon or sinew is a tough band of fibrous connective tissue that connects muscle to bone and is capable of withstanding tension.
Tendons are similar to ligaments; both are made of collagen. Ligaments join one bone to another bone, while tendons connect muscle to bone.
Tendon inflammation results in degeneration or weakening of the tendons, which eventually lead to tendon rupture
WHEN YOU JOIN A KALARI SCHOOL AT A YOUNG AGE, THE FIRST YEAR GOES IN STRENGTHENING TENDONS AND FOOT MASSAGES
A tendon rupture is THE most serious problem for an athlete and will result in excruciating pain and permanent disability if untreated.
Tendon rupture occurs in a man over the age of 60. In the young men of age less than 30 , the muscle usually tears before the attached tendon does.
As you age, your blood supply decreases. This decreases blood going to the tendon, resulting in weakening of the tendon.
Tendons are the sinew that connect muscles to bones and then transmit force from your muscles to your bones
Tendons are similar to ligaments; both are made of collagen.
Ligaments aren’t muscles so they can’t be “worked out” and strengthened easily.
Tendons connect muscles to bones and allow the body to move; ligaments connect bones to bones and protect and stabilize joints such as ankles, knees, hips, wrists and shoulders. Healthy tendons and ligaments provide an athlete with the strength and agility required for high-impact sports.
Given their lack of direct blood supply, these tissues are very slow to heal. Thus it is best to avoid injuring them in the first place.
Again, ligaments connect bone to bone and tendons connect muscle to bone
Each muscle has tendons attached at each end. Tendons are designed to only stretch a small amount. Their job is to transmit force between the bones and the muscles.
For example, when the biceps muscle on the front top of the arm contracts, the tendon attached to the biceps muscle and elbow bone helps the muscle to pull on the elbow bones so the joint can bend.
When properly developed, a tendon has good elasticity and is strong and capable of great power. Tendons essentially cause the bone to move as they transmit tensile load produced by the muscles
A tendon is flexible, tough and fibrous and it can withstand tension.
Tendons and muscles work together and exert a pulling force. Tendons and ligaments are tough and fibrous, but they are known as soft tissue, because they are soft compared with bone.
Without tendons, you wouldn’t be able to control the movement of your body.
A protective sheath known as the synovium covers tendons. This sheath produces synovial fluid, which keeps the tendon lubricated. Injury to the tendon may result in the malfunction of the sheath. If this occurs, the sheath may fail to make synovial fluid or it may not make enough fluid.
This can cause inflammation or swelling of the sheath. This condition is known as tendon sheath inflammation. It’s also sometimes called tenosynovitis.
A synovial sheath is one of the two membranes of a tendon sheath which covers a tendon. The other membrane is the outer fibrous tendon sheath.
The tendon invaginates the synovial sheath from one side so that the tendon is suspended from the membrane by the mesotendon, through which the blood vessels reach the tendon, in places where the range of movement is extensive.
The mesotendon disappears or remains in the form of narrow tendinous bands as threads known as vincula tendina.
The synovial sheath is found where the tendon passes under ligaments and through osseofibrous tunnels; their function is to reduce friction between the tendon and their surrounding structure.
They lie internal to the fibrous tendon sheaths . The synovial membrane makes synovial fluid, which has a lubricating function. The synovial fluid, keeps the tendon moist and lubricated. Tendon sheaths consist of two layers: a fibrous layer, made of tight collagenous tissue, and a synovial layer.
A tendon sheath is a layer of synovial membrane around a tendon. It permits the tendon to stretch and not adhere to the surrounding fascia. The tendons receive their blood supply through synovial folds known as vincula, each tendon having two, vincula longa and vincula brevia.
Tendons become less flexible with age and more susceptible to injury.
Tendons are designed to withstand bending, stretching, and twisting, but they can become inflamed from traumatic injuries that leave them with torn fibers or other damage (tendinitis) or, more commonly, fail to heal or scar following overuse (tendinosis)
Your tendons are designed to withstand high, repetitive loading, however, on occasions, when the load being applied to the tendon is too great for the tendon to withstand, the tendon begins to become stressed.
Tendon deteriorates with age, and disuse, which can be mitigated at least in part by resistance exercise..
A chain is ONLY as strong as as it's weakest link and chances are your weak link are your tendons and ligaments. Most power lifters have failed to strengthen the connection of their muscle to their bones and their bones to each other.
By strengthening these connective tissues you can unlock the true potential of the muscles, who are shackled by the limits of the tendons. Most of the time life changing injuries are caused by a tendon or ligament tear.
Tendons and ligaments are essential in terms of resistance training and the cultivation of physical power. The tendon connects muscle to bone like a thick securing strap, that also can act like a muscle aiding in the contraction and extension of a joint, while stabilising it and making sure that joint doesn't slip out of place or that muscle tear off the bone.
With proper training and development the tendons developed great elasticity and incredible power. It is the tendon that transmit load from the muscles to the bones and moves the body. And building your muscles too fast - without the training of the tendons, leave you without the full ability to call upon all your strength resources while leaving you susceptible to injury.
The ligaments meanwhile are tough cord-like fibres with greater flexibility tying and binding our bones together at joints and allowing for movement in a specific direction.
If you fail to strengthen these connective tissues, you're looking at serious injury with the typical tendon rupture taking more than a year to recover from.
Isometrics for strengthening tendons and ligaments is the best way to build strength and prevent injury. By training the muscle and tendon's side by side in a progressive program you ensure you muscle and never limited by weak connections, and you muscles aren't threatened by overly tight tendons.
ISOMETRIC EXERCISES STRENGTHEN TENDONS ..
YOGA AND KALARI USED ISOMETRICS AND GRAVITY.
YOGIS AND KALARI FIGHTERS HAD LEAN MUSCLES BUT WERE TWICE A STRONG AS BULKY ISOTONIC PROFESSIONAL BODY BUILDERS LIKE ARNOLD SHAWRAZNEGGER WHO USED GYM MACHINES .
During exercise, muscles can develop tension while shortening, lengthening or staying the same length. Muscle shortening, known as concentric contraction, forces a joint angle to decrease. Muscle lengthening, known as eccentric contraction, causes a joint angle to increase. When tension develops in a muscle but the length does not change, the joint does not move, and the contraction is said to be isometric.
When comparing isotonic to isometric exercise, you are comparing exercises that respectively initiate joint movement to exercises that are static, causing no movement. Isometric contractions are muscle contractions where there is no change in the length of the muscle. The contractions occur because the force exerted by the muscle contraction is only equal to the opposing external force, such as gravity.
Isometric exercises are static, meaning no joint movement is involved. The training effect of isometric exercise is specific to the joint angle at which it is performed, within five to 25 degrees.
Yielding isometric means that the lifter applies only enough pressure to the weight to cancel the resistance, causing the weight to be neither dropped or lifted. Overcoming isometric means that no matter the amount of pressure applied by the lifter, the weight simply does not move. Overcoming is a more pure isometric, due to the fact that the weight is an immovable force.
BELOW: THE MOST POTENT EXERCISE ON THIS PLANET FOR BODY MIND AND SPIRIT-- SURYA NAMASKAR
THE POST BELOW IS THE MOST WIDELY READ AMONG MY 1276 POSTS -- 96% READERS ARE FOREIGNERS
Connective tissues have very poor blood flow compared to their adjacent muscles. A tendon gets something like 2% of the blood moving through a nearby muscle while at rest, and that only increases by around seven times during activity -- whereas a muscle will see more than 20 times the flow as at rest.
Tendons are living tissue and respond to mechanical forces by changing the metabolism as well as their structural and mechanical properties. For example, tendons exhibit increase cross sectional area and tensile strength, with increased tendon fibroblast production of collagen type I in response to appropriate physical training.
However, inappropriate physical training leads to tendon overuse injuries or tendonopathy and excessive repetitive stretching of tendon fibroblast increases the production of inflammatory mediators such as prostaglandin E2 and leukotriene B4.
The ability of connective tissues like tendons to alter their structure in response to mechanical loading is referred to as tissue mechanical adaptation. Forces generating muscles are transmitted to bone through tendons, which makes joint and limb movement possible.
To do this effectively, tendons must be large forces, but the level of muscle contraction and the tendons relative size influence mechanical forces on the tendon. In general, the greater the cross sectional area of the muscle, the higher force it produces and the larger stress a tendon undergoes.
Different activities induce different levels of forces even on the same tendon and varying the rate and frequency of mechanical loading results in different levels of tendon forces.
Because of their elasticity, tendons are more deformable at low strain rates. Therefore, the tendons absorb more energy but are less effective in transferring loads. At high strain rates, tendons become less deformable with a high degree of stiffness and are more effective in moving large load
As we age, our tendons get stiffer, collagen turnover is way down -- so wear-and-tear can add up as healing is slowed
The dry weight of each wall of tendons is made up of more than 95% of collagen. The ends of tendons, which are the most solid parts, are composed almost exclusively of collagen, up to 99%. Our tendons’ properties and functions are directly related to the architecture and quality of the collagen fibres.
Tendons provide an essential support in joint movement. The multiple functions of tendons include attaching muscles to bones, joining and stabilizing skeletal joints, and transmitting muscular force to bone pieces. Tendons also allow muscles to be at an optimal distance from the joint.
Tendons, together with muscles and bones, form a unique team that constantly works in synergy. The decrease of collagen with aging weakens the area where tendons are attached to the bone. This creates a more fragile area and frequently causes several injuries.
KAPIL DEV WAS ONE FAST BOWLER WHO HAS ALMOST ZERO INJURIES .. HE IS A UNSUNG HERO OF INDIA
Again, ligaments join one bone to another bone, while tendons connect muscle to bone.
Power lifting principles focus on developing tendon and ligament strength to lift the weights they lift
The true secret to strength, lies in the deliberate strengthening of the ligaments and tendons, by isometric exercise
Because of its ample blood supply, high metabolic activity and ability to contract and stretch, muscle tissue can recover from and adapt to such training quickly. In contrast, tendons and ligaments are more rigid, less metabolically active and have a comparatively low blood supply.
When a bodybuilder cranks up his training to the nth degree, the muscles might be able to withstand forces that the connective tissues cannot.
Power lifters and power bodybuilders y need to focus on developing tendon and ligament strength to lift the weights they lift without long injury layoffs
Ligaments are made of the same material as tendons. Ligaments connect the bones to each other, and are designed to help stabilize the joints and provide a structure for the bones.
Since they have limited stretching ability, they limit how far a joint moves to help protect against injury. As the elbow joint bends, the ligaments stabilize the elbow bones so the arm can move with control.
A sprain is a stretch or tear in a ligament.
A strain is also a stretch or tear, but it happens in a muscle or a tendon.
Ligaments are tough cord-like fibres with greater flexibility.
Ligaments mediate the guidance of joints and skeletal elements. They are coarse, fiber-rich connective tissues that connect different skeletal elements and have mainly stabilizing functions. The histological structure of the ligament is very similar to the structure of tendons.
Inflammatory processes, like in rheumatoid arthritis , can cause a chronic damage to the tendons, tendon sheaths, the articular capsule, and the ligaments, thus deforming the joints .
Tendons transfer forces from muscle to bone to produce joint motion . Tendons orient themselves along stress
Tendons contain more collagen and are less viscoelastic than ligaments
Collagen provides strength and stiffness while elastin allows the joint to extend. Given the vitalness of tendons and ligaments in terms of training with maximal weights, it is important to strengthen them
Collagen is a protein with a high tensile strength, providing structure and elasticity to skin, tendons, ligaments and bones. It is the most abundant protein in the body.
Tendons consist of collagens, proteoglycans, glycoproteins, water and cells. Tendons are rich in collagens with the most abundant tendon component being type I collagen, constituting about 60% of the tendon and about 95% of the total collagen.
The remaining 5% consist of type III and V collagens. In normal tendons, type III collagen is mainly located in the endotenon and epitenon. However, it is also found in aging tendons and at the insertion sites of highly stressed tendons such as the supraspinatus. Type III collagen forms smaller less well organised fibrils and type I collagen, which may result in decreased mechanical strength.
Tendons and ligaments are tough, fiber-rich connective tissues characterized by their excellent tensile strength he collagen fibrils consist primarily of collagen type I and very small amounts of elastin and other collagens
Tendons are responsible for the power transmission as well as for the stabilization of joints and skeletal elements. In tendons the collagenous fibers, which are parallel and oriented in tensile direction, are divided by septa of loose connective tissue (peritendineum) to separate bundles.
On the outside, the tendon is enveloped by a white fibrous sheath called epitendineum, which merges into the perimysium, the connective tissue surrounding the muscle.
Most anabolic steroids reduce extracellular water, which impact tendon stiffness and flexibility..
Like any protein, collagen is made up of long chains of amino acids, however the high concentration of proline and glycine make collagen quite unique.
Collagen often forms the backbone, onto which those other components attach, e.g. minerals such as calcium, phosphorus and magnesium in bones.
Common signs of weak connective tissue and decreased collagen synthesis:-----
-Decreased skin elasticity, sagging, wrinkles, easy bruising
-Joint problems such as arthritis, stiffness and pain
-Frequent sport injuries e.g. torn ligaments
-Broken facial capillaries - so called ‘spider veins’
-Bone problems – osteopenia, osteoporosis, fractures
Collagen synthesis generally decreases with age.
Type I collagen forms fibers and is found in most connective tissues with bone, ligaments, tendon and skin having the high concentrations of type I collagen.
Type II collagen forms fibers but are less well organized than type I fibers. They are found mainly in cartilage.
Type III collagen forms fibers but these fibers are much thinner than type I. Type III collagen make up reticulin fibers in organs and help organize cells within organs.
Type IV collagens form branched networks and help organize the basement membrane.
Type I collagen makes up 85% of dry weight of tendons. Type III collagen make up 0-5% of dry weight of tendons
Collagen types contain different proteins which serve separate purposes within the body. Types 1 & 3 can be taken together and may support skin, muscles, bone health, and hair and nail growth and maintenance
Collagen Type 2 protein makes up the fluids and function in the cartilage and joints.
There are 19 amino acids (proteins) found in Collagen Types 1 & 3 – each essential to functions and maintenance within skin, muscles, and bones. These types are produced by fibroblasts (cells in connective tissues) and osteoblasts (cells that make bones).
Over 90 percent of collagen in the body is comprised of Type 1 & 3 Collagen. Proteins in these types include glycine, proline, alanine, and hydroxyproline.
Glycine is the amino acid found at the highest levels in collagen, requires more serine than our bodies can naturally produce. Studies suggest the need for glycine supplementation to achieve ideal metabolic processes
Proline is a non-essential amino acid synthesized from glycine and important in function of joints and tendons
Hydroxyproline is an amino acid that plays an essential role in the stability of collagen
Alanine is an amino acid used in the biosynthesis of proteins
Collagen Type 2 is produced by chondrocytes (the non-cellular matrix of cartilage) – a liquid-like filling within the cartilage.
Over time, the ideal levels of collagen our bodies naturally produce begin to wane. Collagen fibers break down or no longer regenerate, and lead to a “deflation” effect on the surface of the skin.
The diminishing effects of reduced collagen Type 2 are less obvious and often go unnoticed or are considered “normal” for the aging process.
By the age of 40, the body’s ability to produce collagen decreases by 28%. By age 60, it has decreased by over 54 %.
Despite being made up of non-essential amino acids, the supply of ideal levels of naturally occurring collagen cannot meet the demand.
Proteoglycans make up 0-5% of dry weight of tendons
Decorin is the most predominant proteoglycan in tendon regulates collagen fiber diameter (length of 300nm, diameter of 1.5nm) forms cross-links between collagen fibers and transfers loads between collagen fibers
Aggrecan is proteoglycan found in areas of tendon compression
Fibroblasts are the cells that make up the structural framework or stroma composed of the extracellular matrix and collagen in animal tissues. These cells are the most common type of connective tissue in animals, and are important for wound healing.
Dense connective tissue is used to form ligaments and tendons. Ligaments are rope-like tissue bundles that attach bones to each other and are found in joints whereas tendons attach bones to the surrounding skeletal muscle tissue.
Dense connective tissue also makes up the dermis. The tissue matrix is made almost entirely of collagen, which is the most abundant protein in mammals. Interspersed between the collagen fibers of dense connective tissue are fibroblasts which produce a collagen subunit, tropocollagen, used to construct larger collagenous aggregates.
Fibroblasts also produce glycoproteins and polysaccharides for the ground substances, a gel-like material that surrounds collagen fibers of dense connective tissue. This forms an “extracellular matrix” which contributes to the structural integrity of ligaments and tendons and determines the physical properties of connective tissue.
In addition, fibroblasts have a tissue repair function and wounds stimulate fibroblast production. Collagen’s ubiquity makes fibroblasts the most common cells of connective tissue in mammals.
Fibroblasts are predominant cell type spindle shaped and arranged in parallel rows in direction of muscle loading They produce mostly type I collagen (85% of dry weight of tendons) and also produce small amount of type III collagen (5% of dry weight) responsible for healing process
If you removed the cells from one of your organs, a collagen 'skeleton' of that organ would be left behind. Collagen is the major constituent of your tendons and ligaments, and much like the tough backing on a carpet, it provides support for your skin.
When you have an injury to your skin, collagen is the stuff that forms the scar. Due to the many tasks required of connective tissue, your fibroblasts 'know' how to make at least a dozen different types of collagen.
Fibroblasts play a critical role in an immune response to a tissue injury. They are early players in initiating inflammation in the presence of invading microorganisms.
Both anabolic steroids and fluoroquinolones cause dysplasia of collagen fibrils, which, in turn, decrease tendon tensile strength . Fluoroquinolones are antibiotics that are commonly used to treat a variety of illnesses such as respiratory and urinary tract infections.
When you lift something heavy, the muscles are transmitting force through the skeleton through all those tendon attachments, and they're stimulated to grow the same way as muscles. Mechanical tension and stretch signals remodelling and protein synthesis, only in tendons it's collagen turnover that's stimulated.
In aging tendons collagen content reduces in an age-dependent manner; that is, collagen synthesis as well as collagen turnover diminish with increasing age
Training induces biomechanical changes in tendons. Strenuous endurance training increases collagen turnover but decreases collegan maturation in tendons. Exercise stimulates tenocytes to increase the expression of Insulin-like Growth Factor 1
IGF-1 is a potent stimulus of collegan synthesis and cell proliferation. As such, the IGF-1 may serve as a protein marker for tendon remodelling activities. This results in a net synthesis of type I collegan production.
There have been few studies to determine the affects of tendon disuse and immobilisation; however, it is known that the affect of disuse and immobilisation on tendons is much slower and less dramatic than on skeletal muscles because they have a much slower metabolism and vascularity.
In general however, immobilisation decreases the total weight of the tendon, stiffness and tensile strength. Immobilisation caused the formation of irregular and uneven collagen fibres, dilated veins and capillaries.
Stress deprivation due to immobilisation is responsible for the degenerative changes in tendons. Tendon healing can be largely divided into 3 overlapping phases, inflammatory repairing and remodelling phases:
The initial inflammatory phase, which lasts about 24 hours, erythrocytes, platelets and inflammatory cells (eg: neutrophils, monocytes and macrophages) migrate to the wound site and clean the site of necrotic materials by phagocytosis. In the meantime, these cells release phaso active and chemo tactic factors which recruit tendon fibroblast to begin collegan synthesis and deposition.
A few days after the injury, the repairing phase begins. In this phase, which lasts a few weeks, tendon fibroblast synthesise abundant collegan and other extra cellular matrix components such as proteoglycans and deposit them at the wound site.
After about 6 weeks, the remodelling phase starts. This phase is characterised by decreased cellularity and decreased collagen and glycosaminoglycan synthesis. During this period, the repair tissue changes to fibrous tissue, this again changes to scar like tendon tissue after 10 weeks.
During the later remodelling phase covalent bonding between the collagen fibres increases resulting in repaired tissue with highest stiffness and tense our strength. Also, both the metabolism of tenocytes and tendon vascularity decline.
During tissue healing growth factors play an important role in this process.
1: Platelet Derived Growth Factor (PDGF) is produced shortly after tendon injury and stimulates the production of other growth factors.
2: TGF-beta is active during the inflammatory and repair phases of tendon healing. TGF-beta plays a major role in the repair of injured tendons. TGF-beta 1 aids an extra cellular matrix deposition; however, it’s over expression results in tissue fibrosis. TGF-beta 2 functions similarly to TGF-beta 1. However, TGF-beta 3 has been shown to improve tissue scarring. Peak levels of TGF-beta receptory expression occur at day 14 post injury and decrease until day 56 post injury.
3. Vascular Endothelial Growth Factor (VEGF) stimulates endothelial cell proliferation, enhances angiogenesis and increases capillary permeability. VEGF RNA expression is detected at the repair site 7 days post injury with peak levels at 10 days post injury.
4. Nitric Oxide Synthase (NOS) isoforms are expressed with differential expression patterns during the 3 phases of tendon healing.
Except for degenerative tendons (tendonosis), injured tendons tend to heal. However, the healing tendon does not reach the biomechanical properties of the tendon prior to surgery.
Collagen is one of the most abundant proteins in the body and it makes up a large part of our skin, hair and nails. Technically a polypeptide, collagen contains a mixture of amino acids like proline and glycine, which are found in all connective tissue within the body (including vital organs!).
VIRGIN COCONUT OIL AND DESI GHEE ( HUMPED COW ) PROMOTES COLLAGEN PRODUCTION.
Coconut oil boosts collagen production, Collagen is the fibrous protein that connects tissue. Collagen, a structural protein in the inner layers of skin, is what keeps the skin elastic and smooth. It may breakdown due to the effect of UV rays, pollution, smoking, and physical injuries.
Replacement of collagen is necessary to counteract the effects of its breakdown. It has been found that coconut oil . Coconut oil also encourages the production of collagen, which some find key to a youthful appearance.
Virgin Coconut Oil (VCO) will help heal inflamed tendons. REM sleep is important
Collagen is the most abundant of your body’s proteins, found in your connective tissues, muscles, bones, tendons, blood vessels and digestive system, and it comprises 72 percent of your skin’s protein
By your mid-20s, the collagen in your body starts to diminish, and by the time you reach your 80s, you have around four times less collagen, which explains sagging skin and wrinkles
Energy storing tendons can store and recover energy at high efficiency.. Tendons have the highest tensile strength of all connective tissue because of a high proportion of collagen in the fibres and their closely packed parallel arrangement in the direction of force.
Depending upon the degree of mineralization, collagen tissues may be rigid (bone), compliant (tendon), or have a gradient from rigid to compliant (cartilage). It is also abundant in corneas, cartilage, bones, blood vessels, the gut, intervertebral discs, and the dentin in teeth.
In muscle tissue, it serves as a major component of the endomysium. Collagen constitutes one to two percent of muscle tissue, and accounts for 6% of the weight of strong, tendinous muscles. The fibroblast is the most common cell that creates collagen.
Bone and tendon contain predominantly collagen fibers to provide robust mechanical strength, whereas the connective tissue in most other organs has fewer collagen fibers and more proteoglycans. Cartilage is a specialized form of connective tissue that is rich in proteoglycans and hyaluronan. Cartilage resists compression and covers the surfaces of most joints.
Loose connective tissue is found in most organs and provides mechanical, metabolic and immune support. Loose connective tissue is highly cellular and rich in proteoglycans; it contains fewer collagen fibers. Loose connective tissue controls the diffusion of metabolites and signaling molecules and contains blood vessels and immune cells.
Dense connective tissue is found in the walls of large arteries, dermal layer of skin, tendon and bone. Dense connective tissue contains fewer cells and more collagen fibers. In tendon and bone, the collagen fibers are arranged in very precise order to provide very robust tensile strength.
Collagens are the main structural component of connective tissue and they resist tensile or stretching forces. Collagens comprise a large family of proteins of around 40 types but there are four types that are most common.
BELOW: THE STRENGTH IN THE VIDEO BELOW COMES FROM HIGH DENSITY BONE AND STEEL WIRE TYPE TENDONS / LIGAMENTS
REAL STRONG MEN DO NOT HAVE A "ONLY FOR SHOW" BODY LIKE ARNOLD SCHWARZENEGGER ..
THEY NEED STRIKE ONLY ONCE ---NOT 100 TIMES LIKE CHOOT MOSQUITO TYPE BRUCE LEE -- HOOO HOOO HOOOO
REAL STRONG MEN HAVE BARREL BODIES --
THE SPINE HAS TO BE ANCHORED WELL
YOU SEE OLYMPIC WEIGHT LIFTING COMPETITIONS -- ALL LIFTERS HAVE BARREL BODIES -- THEIR SIX PACKS CANT BE SEEN
BELOW: ZYDRUNAS SAVICKAS TORE HIS TENDONS / LIGAMENTS DURING THIS CONANs WHEEL CONTEST IN 2010 AT FAROE ISLANDS .
PAIN IS AN INDICATION FROM BRAIN-- STOP NOW !!
The Achilles tendon is a strong fibrous cord that connects the muscles in the back of your calf to your heel bone. Have you seen how many athletes rupture their Achilles tendon ? These athletes have been fooled into injecting steroids into their tendons.
Read all 4 parts of the post below--
The Achilles tendon is a strong fibrous cord that connects the muscles in the back of your calf to your heel bone. If you take anabolic steroids and overstretch your Achilles tendon, it can tear (rupture) completely or just partially.
Fluoroquinolone antibiotics, such as ciprofloxacin (Cipro) or levofloxacin (Levaquin), increase the risk of Achilles tendon rupture.
If your Achilles tendon ruptures, you might hear a pop, followed by an immediate sharp pain in the back of your ankle and lower leg that is likely to affect your ability to walk properly
Doctors sometimes inject steroids into an ankle joint to reduce pain and inflammation. However, this medication can weaken nearby tendons and has been associated with Achilles tendon ruptures.
Chutney Marys ape western women and wear high heels. If you wear high heels daily, youwill find that your Achilles tendon at the back of your ankle changes, becoming tight, thickened and shortened.
The strain and pain of high heels doesn't stop at your feet. It can creep all the way up into your back. High heels tend to push your body weight forward. This means you need to work harder to prevent yourself from falling forward.
This can place extra strain on your knees, and can alter the natural curvature of your back, which can lead to knee and back pain. The joints of the feet can be damaged by wearing high heels, and this can cause deformities and some forms of arthritis.
A two inch heel increases the pressure on the ball of your foot by about 52 per cent with each step you take. Wear a heel over three inches (8cm) and the pressure increases to about 79 per cent. What does that do to your foot? Unnatural loading of the forefoot can lead to hammer toe deformities, bunions, callouses and thickening of the nerves between the toes
Tendonitis, is the inflammation of a tendon. It happens when a person overuses or injures a tendon, for example, during sport. It is normally linked to an acute injury with inflammation.
If the sheath around the tendon becomes inflamed, rather than the tendon itself, the condition is called tenosynovitis. TendOnitis and tenosynovitis can occur together.
Sometimes your doctor may inject a corticosteroid medication around a tendon to relieve tendinitis. Injections of cortisone reduce inflammation and can help ease pain. Corticosteroids are not recommended for tendinitis lasting over three months (chronic tendinitis), as repeated injections may weaken a tendon and increase your risk of rupturing the tendon.
Typically, tendon injuries occur in three areas:--
musculotendinous junction (where the tendon joins the muscle)
mid-tendon (non-insertional tendinopathy)
tendon insertion (eg into bone)
The inability of your tendon to adapt to the load quickly enough causes tendon to progress through four phases of tendon injury. While it is healthy for normal tissue adaptation during phase one, further progression can lead to tendon cell death and subsequent tendon rupture.
Up to 35% of all body protein content is made up of one type of protein, collagen. This protein is a key component of our connective tissue – the most widely distributed tissue in the body, which binds, holds, supports and strengthens other body tissues, as well as insulating and protecting the organs.
It makes up our ligaments, tendons, skin, joints, cartilage, bone and even the gut lining, blood vessels and our teeth! It is therefore absolutely vital to health and proper integrity and function on ALL body tissues and organs.
If you have an inflammatory condition, there is a higher chance of collagen damage
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With supraspinatus tendinitis, the tendon around the top of the shoulder joint becomes inflamed, causing pain when the arm is moved, especially upwards.
Some patients may find it painful to lie on the affected shoulder at night. If other tendons in the same area are also affected, the patient may have rotator cuff syndrome.
Your rotator cuff is located in the shoulder and is actually composed of 4 muscles that function together to raise your arm out to the side, to help you rotate the arm, and to keep your shoulder from popping out of its socket.
The rotator cuff tendon is one of the most common areas in the body affected by tendon injury. Some studies of people after death have shown that more than 22% have rotator cuff tears.
Rotator cuff tendon rupture happen when you lift a heavy object overhead repeatedly.. You will be unable to bring your arm out to the side.
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The biceps muscle of the arm functions as a flexor of the elbow. This muscle brings the hand toward the shoulder by bending at the elbow.
Ruptures of the biceps are classified as proximal (close) or distal (far). Distal ruptures are rare. The proximal rupture occurs where the biceps attaches at the top of your shoulder.
Eccentric loading---when your muscle contracts while it is being stretched in the opposite direction, increased stress is placed on the involved tendon.
You will have decreased strength of elbow flexion and decreased ability to raise the arm out to the side when the hand is turned palm up.
If your biceps tendon is completely ruptured, the biceps retracts toward the elbow causing a swelling just above the crease in your arm. This is called the Popeye deformity.
There are three main types of muscles: skeletal muscles, which can be voluntarily controlled, involuntary smooth muscles, such as those that control breathing, digestion and other functions, and involuntary cardiac muscles, which control the function of the heart.
Skeletal muscles travel across the length of joints and stretch between the bones. All muscles in the body contract or shorten when they receive nerve signals initiated by the brain.
All voluntary movement starts with an electrical impulse in the brain. Never try to fool the brain with props.
The brain sends signals via the nervous system that make the appropriate muscles respond. Some muscles contract and shorten, other muscles relax and still others stabilize the movement. As a muscle contracts, the force travels through the muscle and out through the tendon, which tightens and pulls on the bones that need to move.
Steroids are a drug, and all drugs have side effects.
Anabolic steroids, also known more properly as anabolic–androgenic steroids (AAS), are steroidal androgens that include natural androgens like testosterone as well as synthetic androgens that are structurally related and have similar effects to testosterone. They are anabolic and increase protein within cells, especially in muscles
Steroid use floods your body with testosterone Because steroids are hormones, steroid abuse has psychological effects.
When you’re consistently dosing your body with massive amounts of synthetic testosterone, your body stops producing testosterone naturally. The main effect of this: Your testicles shrink and you become infertile--steroids are essentially male contraception.
While natural production will eventually start again if you stay off of steroids, returning to normal can take years
Anabolic-androgenic steroids are a synthetic version of the testosterone your body already makes. They look and act a lot like natural testosterone. The hormone attaches to the receptor sites on our cells and affects your systems just as the natural hormone would
Anabolic-androgenic steroids are the most widely used appearance- and performance-enhancing drugs in the WORLD and are used by everyone from professional athletes to amateurs and adolescents
Anabolic steroids are considered a schedule III drug, so without a prescription, they’re considered illegal in America. Steroid use is banned in most sports competitions, like the Olympics, even if athletes have a ‘script. Majority of American men who take non-prescription steroids are probably getting their steroids through the Internet ( dark web ) via international sellers.
Anabolic steroids are synthetic androgens. Synthetic androgens help men build muscle more easily, burn more fat
Steroids weaken the myocardium -- the middle muscle layer of the heart wall -- leading to cardiovascular complications
Steroids affect the musculoskeletal system as well, increasing the possibility of tendon rupture because of rapid muscle gain
Steroids cause osteoporosis--aa weakened skeletal system makes for a weakened musculoskeletal system. The drug may also weaken the immune system, opening the doors to a long list of muscle-debilitating illnesses or diseases.
Corticosteroids may mask painful symptoms, causing the individual to overexert a weakened tendon.
Corticosteroid injections cause bone weakening (osteoporosis) or bone death (osteonecrosis) and tendon weakening or rupture. Steroid injections cause a deterioration of cartilage in the joint Doctors sometimes inject steroids into an ankle joint to reduce pain and inflammation.
Cortisone is not the same as the illegal steroid drugs taken by athletes and bodybuilders.
Too much steroid use can weaken tendons so much that one could ﬂex and the tendons rip and rupture. It then requires rather invasive surgery to repair and reattach the ruptured tendons. In rare cases if a tendon goes untreated it can cause permanent disability and chronic pain.
Byproduct of steroid use is an increase in aggression or even rage known as “Road Rage” the anger and potential outbursts are often attributed to excessive testosterone levels found in long time users.
Elevated levels can amplify already existing personality traits or conditions resulting in aggressive outbursts.
Without tendons, you wouldn’t be able to control the movement of your body.
A protective sheath known as the synovium covers tendons. This sheath produces synovial fluid, which keeps the tendon lubricated.
Tenosynovitis is the inflammation of the fluid-filled sheath (called the synovium) that surrounds a tendon, typically leading to joint pain, swelling, and stiffness. Tenosynovitis can be either infectious or noninfectious.
The most common manifestation of infectious tenosynovitis is in the flexor tendons of the fingers..
Injury to the tendon may result in the malfunction of the sheath. If this occurs, the sheath may fail to make synovial fluid or may not make enough fluid.
This can cause inflammation or swelling of the sheath. This condition is known as tendon sheath inflammation. It’s also sometimes called tenosynovitis.
Tendon sheath inflammation is typically the result of injury ( STRAIN ) to the tendon or surrounding muscle or bone. Some people may develop a fever. This indicates the presence of an infection and requires immediate medical attention.
Tendons are thick cords that join your muscles to your bones. When tendons become irritated or inflamed, the condition is called tendinitis. Tendinitis causes acute pain and tenderness, making it difficult to move the affected joint.
Enthesitis is inflammation of the entheses, the sites where tendons or ligaments insert into the bone. It is also called enthesopathy, or any pathologic condition involving the entheses.
Tendons are the tissues that attach your muscles to your bones. Ligaments are what attach your bones to one another.
The places where your tendons and ligaments meet your bones are called entheses. Sometimes, these connection points can get inflamed and become painful because of injury, overuse, or disease. This is known as enthesitis, also sometimes called enthesopathy.
Inflammation of the entheses can cause new bone tissue to form. That new bone tissue gets in the way of normal movement and function -- like a bone spur on your heel.
Common spots for enthesitis to happen are around your heel, knee, hip, toes, fingertips, elbow, backbone, and the bottom of your foot. You'll feel pain and stiffness, especially when you're moving. You also might notice swelling around those areas.
As the condition gets worse, it can make your tendons and ligaments thick, swollen, or hard, and that makes movement even more painful and difficult. These changes can often be seen with an imaging test such as an ultrasound or MRI.
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SHALLOW PETTY MINDS ( LIKE OF BR AMBEDKAR / EVR PERIYAR ) WILL NEVER UNDERSTAND SANATANA DHARMA
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CONCH IS THE GOLDEN SPIRAL
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